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er stress inhibitor tauroursodeoxycholate  (MedChemExpress)


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    MedChemExpress er stress inhibitor tauroursodeoxycholate
    AP-mediated signaling ameliorates ALI by blocking ER stress. The effect of LV-NOTCH1 OE injection and <t>TUDCA</t> on the expression of NOTCH1 ( A ), HES1 ( B ), AK2 ( C ), CHOP ( D ), and GRP78 ( E ) in mouse liver tissues was examined using IHC staining ( n = 5). ( F ) Structural changes in liver pathology caused by LV-NOTCH1 OE injection and TUDCA detected by HE staining ( n = 5). ( G ) Effect of overexpression of NOTCH1 and blockade of ER stress pathway on apoptosis in the liver of mice detected by TUNEL ( n = 5). Data are mean ± SEM, * p < 0.05, ** p < 0.01, *** p < 0.001, **** p < 0.0001, ## p < 0.01, ### p < 0.001, #### p < 0.0001 by one-way ANOVA followed by Tukey’s test
    Er Stress Inhibitor Tauroursodeoxycholate, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 96/100, based on 116 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/er+stress+inhibitor+tauroursodeoxycholate/pmc11889056-52-1-7?v=MedChemExpress
    Average 96 stars, based on 116 article reviews
    er stress inhibitor tauroursodeoxycholate - by Bioz Stars, 2026-08
    96/100 stars

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    1) Product Images from "Andrographolide ameliorates sepsis-induced acute liver injury by attenuating endoplasmic reticulum stress through the FKBP1A-mediated NOTCH1/AK2 pathway"

    Article Title: Andrographolide ameliorates sepsis-induced acute liver injury by attenuating endoplasmic reticulum stress through the FKBP1A-mediated NOTCH1/AK2 pathway

    Journal: Cell Biology and Toxicology

    doi: 10.1007/s10565-025-10007-9

    AP-mediated signaling ameliorates ALI by blocking ER stress. The effect of LV-NOTCH1 OE injection and TUDCA on the expression of NOTCH1 ( A ), HES1 ( B ), AK2 ( C ), CHOP ( D ), and GRP78 ( E ) in mouse liver tissues was examined using IHC staining ( n = 5). ( F ) Structural changes in liver pathology caused by LV-NOTCH1 OE injection and TUDCA detected by HE staining ( n = 5). ( G ) Effect of overexpression of NOTCH1 and blockade of ER stress pathway on apoptosis in the liver of mice detected by TUNEL ( n = 5). Data are mean ± SEM, * p < 0.05, ** p < 0.01, *** p < 0.001, **** p < 0.0001, ## p < 0.01, ### p < 0.001, #### p < 0.0001 by one-way ANOVA followed by Tukey’s test
    Figure Legend Snippet: AP-mediated signaling ameliorates ALI by blocking ER stress. The effect of LV-NOTCH1 OE injection and TUDCA on the expression of NOTCH1 ( A ), HES1 ( B ), AK2 ( C ), CHOP ( D ), and GRP78 ( E ) in mouse liver tissues was examined using IHC staining ( n = 5). ( F ) Structural changes in liver pathology caused by LV-NOTCH1 OE injection and TUDCA detected by HE staining ( n = 5). ( G ) Effect of overexpression of NOTCH1 and blockade of ER stress pathway on apoptosis in the liver of mice detected by TUNEL ( n = 5). Data are mean ± SEM, * p < 0.05, ** p < 0.01, *** p < 0.001, **** p < 0.0001, ## p < 0.01, ### p < 0.001, #### p < 0.0001 by one-way ANOVA followed by Tukey’s test

    Techniques Used: Blocking Assay, Injection, Expressing, Immunohistochemistry, Staining, Over Expression, TUNEL Assay



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    Image Search Results


    AP-mediated signaling ameliorates ALI by blocking ER stress. The effect of LV-NOTCH1 OE injection and TUDCA on the expression of NOTCH1 ( A ), HES1 ( B ), AK2 ( C ), CHOP ( D ), and GRP78 ( E ) in mouse liver tissues was examined using IHC staining ( n = 5). ( F ) Structural changes in liver pathology caused by LV-NOTCH1 OE injection and TUDCA detected by HE staining ( n = 5). ( G ) Effect of overexpression of NOTCH1 and blockade of ER stress pathway on apoptosis in the liver of mice detected by TUNEL ( n = 5). Data are mean ± SEM, * p < 0.05, ** p < 0.01, *** p < 0.001, **** p < 0.0001, ## p < 0.01, ### p < 0.001, #### p < 0.0001 by one-way ANOVA followed by Tukey’s test

    Journal: Cell Biology and Toxicology

    Article Title: Andrographolide ameliorates sepsis-induced acute liver injury by attenuating endoplasmic reticulum stress through the FKBP1A-mediated NOTCH1/AK2 pathway

    doi: 10.1007/s10565-025-10007-9

    Figure Lengend Snippet: AP-mediated signaling ameliorates ALI by blocking ER stress. The effect of LV-NOTCH1 OE injection and TUDCA on the expression of NOTCH1 ( A ), HES1 ( B ), AK2 ( C ), CHOP ( D ), and GRP78 ( E ) in mouse liver tissues was examined using IHC staining ( n = 5). ( F ) Structural changes in liver pathology caused by LV-NOTCH1 OE injection and TUDCA detected by HE staining ( n = 5). ( G ) Effect of overexpression of NOTCH1 and blockade of ER stress pathway on apoptosis in the liver of mice detected by TUNEL ( n = 5). Data are mean ± SEM, * p < 0.05, ** p < 0.01, *** p < 0.001, **** p < 0.0001, ## p < 0.01, ### p < 0.001, #### p < 0.0001 by one-way ANOVA followed by Tukey’s test

    Article Snippet: The ER stress inhibitor tauroursodeoxycholate (TUDCA, HY-19696, MedChemExpress, Monmouth Junction, NJ, USA) was added to drinking water at a dose of 0.5 g/kg/day (same time as AP treatment) (Qin et al. ).

    Techniques: Blocking Assay, Injection, Expressing, Immunohistochemistry, Staining, Over Expression, TUNEL Assay